After exposure to the spike protein of the COVID-19 virus (e.g., through infection or vaccination), various immunological processes are triggered in the body. This can lead to inflammation in nerve tissue and blood vessels, increased oxidative stress, and reduced oxygen supply to the cells. As a result, cellular energy production in the mitochondria and the oxygen supply to the cells can be negatively affected.
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Symptoms experienced during an acute SARS-CoV-2 infection. Typical symptoms may include fever, cough, fatigue, or respiratory symptoms.
These symptoms are directly related to the acute illness.
The term “Long COVID” is often used when symptoms persist or newly appear more than four weeks after an infection.
Common symptoms include fatigue, breathing difficulties, and trouble concentrating.
The term “Post-COVID syndrome” is generally used when symptoms persist more than three months after an initial infection and cannot be explained by other causes.
Those affected often report persistent physical or cognitive limitations in their daily lives.
Research on Long COVID and Post-COVID in recent years has clearly demonstrated just how complex the underlying biological processes can be. In particular, changes in energy metabolism, oxidative stress, inflammatory responses, and tissue oxygenation are the focus of numerous scientific studies.
SANOPAL closely follows scientific developments in these areas and supports professional exchange on current research findings. The organization prioritizes presenting scientific information in an accessible way for those affected, their families, and healthcare professionals.
COVID-19
In a COVID-19 infection, the SARS-CoV-2 virus invades the cells of the respiratory tract and triggers a series of immune responses. In post-COVID and long COVID, symptoms persist and both physical and mental functions are impaired. This leads to increased oxidative stress and the spread of the virus throughout the body. Studies show that during an acute infection, it is essential to both fight the virus and protect the mitochondria.
Studies describe how the spread of the spike protein in the body can be influenced or accelerated by furins (proteases). Furin production in the body can be increased by hypoxic stimuli (stress), which are associated with HIF-1alpha regulation. Alpha-ketoglutarate is an important cofactor for HIF-1alpha regulation.
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Austrian medical professionals have published their observations and scientific insights regarding spike protein-associated symptoms in a research article. The article examines possible connections between energy metabolism, oxygen supply, and regulatory metabolic processes, and contextualizes these findings within the current scientific discourse.
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